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In fibroblast-centric models, readouts consistent with collagen and matrix component regulation have been reported, supporting the use of GHK-Cu as a laboratory tool in matrix biology research

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Mechanism of action: 1) Activation of amylin receptors (AMY1, AMY2, AMY3 heterodimers of calcitonin receptor with RAMP1/2/3) in area postrema and NTS appetite suppression and enhanced satiation