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Toxin A binds to mucosal receptors and causes cytotoxicity by disrupting cytoplasmic microfilaments and inducing apoptosis

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Such modifications can result in loss of enzymatic activity, altered cellular signaling, and degradation of structural proteins, contributing to cellular dysfunction and death [36, 37]

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Tryptophan catabolites from microbiota engage aryl hydrocarbon receptor and balance mucosal reactivity via Interleukin-22