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High levels of intracellular cysteine promote oxidative DNA damage by driving the fenton reaction

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4j): In the first step, lactate oxidation drives the reduction of NAD+ bound to LDHB and CoQ, resulting in the release of pyruvate, generating NADH/PQQ bound to LDHB

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Expression in RBCs versus liver is due to differential use of tissue-specific promoters, which drive expression as well as tissue-specific exon usage (use of exon 1 but not exon 2 in RBCs and exon 2 but not exon 1 in liver)