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Interactions between these cells with a myriad of growth factors and cytokines derived from vitreous contact and breach of the bloodretinal barrier trigger a cascade of downstream cellular processes, including EMT, chemotaxis, proliferation, excessive extracellular matrix deposition, and cellular migration, leading to the formation of contractile fibrotic membranes and the overt loss of the retinal architecture [280]

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For the head-to-head against the closest comparator, see the Matrixyl vs GHK-Cu skin peptide comparison