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Thus, platelet activation may be associated with several factors, including abnormal RBCs, RBC exposed phophotidylserine, micropartcles, hemolysis, iron overload and ROS resulting in chronic platelet activation and hypercoagulability in -thalassemia/HbE patients (Fig
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Takeaway: Collagen supports tissue building blocks, while BPC-157 is proposed to influence healing signals directly
482 DOX can directly bind Topoisomerase II beta in cardiomyocytes, causing DNA double-strand breaks
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