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Lipolytic actions of leptin are dependent on the leptin receptor, since obese fa/fa Zucker rats (112) and db/db mice (32) that have an inactivating mutation of the long form of the leptin receptor are resistant to leptin-induced lipolysis

Further, targeted metabolomics and isotope tracing experiments uncovered GGCT as a metabolic orchestrator to prioritize Gln to GSH flux for antioxidant defense, while activating glucose (Glc)-fueled anaplerosis to replenish TCA intermediates to fuel proliferation

further demonstrated that SLC25A39 and SLC25A40 cooperatively regulate mitochondrial GSH transport, with SLC25A40 expression influenced by Toll-like receptor 4 (TLR4) and receptor for advanced glycation end products (RAGE) signaling pathways 18

Injections produce faster, more reliable results than oral forms for treating true deficiency