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Sometimes referred to in pharmaceutical literature as emideltide, it was the first substance ever extracted from brain tissue specifically because of its observed sleep-promoting properties

Stability after reconstitution

IV hydration and oral supplements aren't competitors they serve different roles in your wellness toolkit

Key Ingredients & their Benefits: Nicotinamide (Vitamin B3) Brightens skin by inhibiting melanin transfer helps with skin lightening Reduces hyperpigmentation effective for dark spot removal Improves elasticity and barrier function supports anti-aging Glycolic Acid Alpha-hydroxy acid (AHA) that exfoliates dead cells promotes brighter skin Stimulates cell turnover fades dark spots Boosts collagen synthesis reduces fine lines ( anti-aging Licorice Extract Contains glabridin inhibits tyrosinase enzyme skin lightening Reduces pigmentation and inflammation helps with dark spot removal Antioxidant properties mild anti-aging support Vitamin E (Tocopherol) Potent antioxidant protects against free radical damage ( anti-aging ) Supports skin healing and moisture retention Indirectly helps maintain even tone but not a strong lightening agent Arbutin Natural derivative of hydroquinone inhibits melanin production ( skin lightening ) Effective in reducing dark spots and uneven tone Gentle compared to hydroquinone, safer for long-tllerm use D-Panthenol (Provitamin B5) Hydrates and soothes improves skin barrier Reduces irritation from actives indirectly supports anti-aging Not a direct lightening agent but enhances overall skin health Full Ingredient list: Sodium Lauryl Ethyl Sulphate Cocamido-propyl Betaine Propylene Glycol Nicotinamide Glycolic acid D-pathenol Licorice extract Dmdm Hyantoin Sodium Hydroxide Vitamin E Arbutin Butylated Hydroxy Toluene Purified water Parfume EDTA 5 in stock We use cookies on our website to give you the most relevant experience by remembering your preferences and repeat visits

This study demonstrated that sequestration of NRF2 by progerin, which results in protein mislocalization and the impairment of NRF2-mediated antioxidant response, contributes to progerin-associated nuclear aging defects [115]