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In OS-induced senescence models, PERK-mediated upregulation of CCAAT/enhancer-binding protein homolog occurs (54)

Preclinical studies in Alzheimers disease models have demonstrated that pharmacological inhibition of ferroptosis with small-molecule inhibitors such as ferrostatin-1 or liproxstatin-1 alleviates lipid peroxidation, neuronal death, and cognitive decline (Bao et al., 2021

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Exposed PS is then bound by both secreted and transmembrane proteins that mediate interactions between the injured distal segment and the regrowing segment, mechanistically mimicking the process of apoptotic cell death 16,18