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It is driven by factors such as vascular endothelial growth factor (VEGF), transforming growth factor beta (TGF), EDN-1, fibroblast growth factor 2 (FGF-2) produced by mast cells, keratinocytes, and fibroblasts, stimulating endothelial cells [153, 154]

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As the main problem lies in the lack of endogenous insulin production, all T1DM subjects will have low portal insulin levels, even when treated with subcutaneous insulin, as this will only increase systemic insulin concentrations