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This upregulation exhausts intracellular GSH, leading to ROS buildup and oxidative DNA damage that can ultimately generate TP53 mutations, a process that is halted when CHAC1 is knocked down (Wada et al., 2018)

10.1016/0011-393x(95)85135-6 [DOI] [Google Scholar] Yazaki Y., Chow G., Mattie M

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