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Cardiovascular diseases (CVDs) are a leading cause of death worldwide, driven largely by conditions like coronary artery occlusion, hypertension, and strokes

To date, the aberrant expression of VRK2 protein has been proven to play a role in the development of various tumors [14,15,16]

In contrast, H 2 O 2 exposure markedly reduced cell viability, which was significantly restored by MAPKAPK2 overexpression

The above studies have shown that down-regulation of RANKL inhibits osteoclastogenesis and reduces bone loss and joint destruction, which is related to the inhibition of NF-B signal activation

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