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Cerebral blood flow obstruction is the primary pathological mechanism of IS, while brain damage persists and continues to progress during reperfusion (78, 79)

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JC virus was detected in CNS tumors, such as glioblastomas

Furthermore, Nalm6 cells lacking FANCB were significantly sensitive to GSH inhibition even in the absence of FA, which might be a consequence of an increase in endogenous free FA, overall suggesting that GSH supply might be fundamental for Fanconi Anaemia patients (Fig