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In the meantime, studies have confirmed that there is a bidirectional regulatory interaction between -Syn and mitochondrial dysfunction, which leads to structural alterations and functional defects in mitochondria (Li H

Its core pathological feature is excessive accumulation of adipose tissue, accompanied by abnormal proliferation, differentiation, and metabolic dysfunction of adipocytes, which in turn induce a series of complications including insulin resistance, nonalcoholic fatty liver disease, and cardiovascular diseases

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