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Research literature has described BPC-157 as interacting with nitric oxide pathways and balancing protective versus damaging nitric oxide related effects in experimental models
In a 2003 in vitro experiment by Khavinson and colleagues, addition of micromolar concentrations of the peptide to cultured human somatic fibroblasts produced a measurable increase in telomerase reverse transcriptase activity and a corresponding extension of telomere length across 1020 passages, with cell cultures continuing to divide beyond the Hayflick limit observed in untreated controls
Examined in oxidative stress response models: Oxidative stress contributes to telomere shortening and cellular damage
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