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This pathway not only suppresses CD8T cell infiltration but also downregulates major histocompatibility complex class I (MHC I) expression and inhibits interferon-gamma (IFN-) signaling, thereby undermining antitumor immune responses at multiple levels [85]

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BPC-157 works primarily through angiogenesis promotion and nitric oxide system modulation, while TB-500 (thymosin beta-4) operates through actin sequestration and cell migration facilitation

Those with active cancer should consult an oncologist, as growth-factor modulation could theoretically influence tumor growth

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