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Enhancement of experimental fracture-healing by systemic administration of recombinant human parathyroid hormone (PTH 1-34) J Bone Joint Surg Am

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[1] At the cellular level, the tendon work is genuinely mechanistic: in cultured rat tendon fibroblasts, BPC-157 accelerated cell outgrowth, improved survival under oxidative stress, and increased migration in a dose-dependent way, apparently through the FAKpaxillin pathway