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Resistance mechanisms vary with the mode of action (MOA) of antifungal drugs

6e), suggesting that FoB with Gclc deficiency experience a higher basal rate of mitochondrial activity compared to control cells

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FIGURE 2 3 Redox dysregulation in disease states 3.1 GSH in peripheral immune cell infiltration Several studies indicate that neuroinflammatory changes associated with neurodegeneration compromise the BBB or its function by disrupting transport systems, facilitating the infiltration of immune cells, and influencing the BBBs role as a signaling interface

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