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The damaging effects of uncontrolled ROS accumulation on DNA, lipids, proteins, and enzymes are thoroughly examined, underscoring their potential to disrupt cellular functions

have shown that NRF2 promotes nucleotide production via upregulation of PPP and glutamine utilization in response to proliferating signals, which in turn accelerates tumor growth [59]

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After stimulation by the relevant signals, RIPK1 undergoes ubiquitination, recruits RIPK3, forms the RIPK1/RIPK3 complex, mediates MLKL oligomerization and forms specific necrosomes in the cytoplasm, thereby leading to pore formation on the plasma membrane [89] with cell swelling and membrane rupture