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Co-treatment with JMR-132 plus cetrorelix induced a significant increase of BPH-1 cells blocked in S-phase plus cells with lower G0/G1 and G2/M DNA content

This section summarizes the evolution of epidemiologic evidence, from 1996 to the present time 275

Exposure to excessive ROS, including free radical superoxide O 2 - , hydrogen peroxide, and hydroxyl radicals, favors oxidative stress, alters mitochondrial Ca 2+ homeostasis, triggers membrane lipid peroxidation and potentially induces nuclear and mtDNA damage [15] .Sustained elevations in intracellular Ca 2+ concentrations ultimately cause neuronal degeneration and cell death by initiating apoptosis [16]

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