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sedebokerense caused 100% of the infection on the host cells of H

Risk factors for mortality in children with diabetic keto acidosis from developing countries

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In a rat model of MCAO, RIPostC reverses GPX4 reduction and ACSL4 overexpression by increasing ketone body production, thereby alleviating nerve damage

Mislocalized TDP-43 in neurons and glial cells disrupts RNA processing and proteostasis, leading to cellular dysfunction that contributes to pathological glial alterations and neurodegeneration