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Recent clinical trials on glutamine metabolism inhibition mainly focuses on using CB-839 to target glutaminase (NCT02071862, NCT02071888 and NCT02071927 for solid tumors, lymphoid, and myeloid malignancies respectively) [24, 62]

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[97] observed that the serum levels of MDA, c-telopeptide degradation products of type II collagen (CTX-II), carboxy propeptide of type II collagen (CPII) were higher in the STR/Ort (Str) mice than in CBA mice

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