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rigorous human clinical trials are limited

Electrophysiology results Based on a well-known phenomenon in peripheral nerve injury (i.e., as the number of preserved motoneurons decreases, the MUP (giant potential) in the tail muscle increases), it is conceivable that the BPC 157-treated rats that underwent spinal cord injury and were subjected to EMG recordings exhibited a markedly lower MUP in the tail muscle than that in the corresponding controls (Table 3)

doi: 10.1172/JCI65907

BPC-157 was first isolated from body protection compound (BPC), which is found in the gastric juices and is part of the digestive process [1]

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