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Both genetic ablation and chemical inhibition of TRF1 telomeric localisation can block the growth of p53-null K-Ras G12V -induced lung carcinomas and PDGFA/B-induced glioblastomas in a telomere-length independent manner, remarkably without affecting mouse survival or tissue function 1,2,3

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Critical roles in angiogenesis (VEGF) and cellular signaling pathways

Blockade of IDO-kynurenine-ahR axis ameliorated colitis-associated colon cancer via inhibiting immune tolerance

There is no current evidence that existing small peptide or non-peptide BDNF mimetics promote survival through inactivation of BAD proteins in various damaging cell context considering the available data