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Accessed August 6, 2012

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Mitochondrial dysfunction initiates intracellular signaling cascades that can impair many processes known to be important in the progression of the disease including autophagy, inflammation, and cellular responses to growth factors

Good connections have been observed between CYP1A protein levels, EROD activity, and research mechanisms of CYP1A-induced toxicity, suggesting that EROD activity not only indicates chemical exposure but also precedes effects at various levels of biological organization (Koglin et al., 2016)