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High insulin levels in the post-prandial state decrease hepatic VLDL production, favoring chylomicron-mediated delivery of dietary lipids to the periphery [111], but the selective hepatic insulin resistance in patients with NAFLD allows insulin to stimulate DNL without inhibiting VLDL production [118]

Cell-cell interactions, particularly involving cancer-associated fibroblasts (CAFs), significantly influence tumor biology, signaling, and metabolic regulation