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Within the H-site, the substrate analogue ( 12 ) interacts through hydrogen bonds to N108 and the backbone of V12, a interaction with F112, water bridges to R16 and Y175, and hydrophobic interactions with V12 and V115 (Fig

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Following treatment with the Gln metabolism inhibitor JHU083, tumor cell growth was suppressed, and the number of CD8+ T cells increased, thereby enhancing the efficacy of the PD-1 blocker (40)

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