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How compartment-specific GSH metabolism modulates GPX4 activity during ferroptotic stress remains an important unresolved question

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This IGF-1-mediated autophagy reduction is suggested to be involved in cellular senescence and longevity

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However, we cannot exclude that commitment of the FoB compartment and the reflection of wild-type MZB-like properties by FoB in B cell-specific Gclc -deficient hosts, might be the consequence of a physiological attempt to fill the empty MZB compartment